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Affected. Kidney weights were increased with 2.1% NH4 Cl. Hypertrophy of the adrenal zona glomerulosa occurred with KHCO3 , KCl and NH4 Cl, due to chronic stimulation of the adrenal cortex by either K + or Cl-induced acidosis. An early onset from week 13 ; of oncocytic tubules was noted in the kidneys of rats fed KHCO3 and, after 30 months, the incidence of this lesion was much higher than the background incidence in ageing controls. No progression to oncocytomas was noted. KCl showed only slight effects on the early onset of oncocytic tubules from 18 months ; . In contrast, the severity of nephrosis and the incidence of oncocytic tubules were decreased with 2.1% NH4 Cl, suggesting a protective effect of acidosis. The feeding of KHCO3 resulted in hyperplasia, papillomas and carcinomas of the urinary bladder. With KCl only a slight increase in proliferative urothelial lesions was noted. Apart from these pre- ; neoplastic lesions in the urinary bladder there were no treatment-related differences in tumour response among the groups. We concluded that most of the observed changes represent physiological adaptations to the feeding of acid- or base-forming salts. Remarkable effects noted with KHCO3 , and to a far lesser extent with KCl, consisted of renal oncocytic tubules and pre- ; neoplastic lesions of the urinary bladder epithelium. NH 4 Cl-induced chronic metabolic acidosis was not associated with dissolution of alkaline bone salts in rats. Finally, a protective effect of chronic acidosis on tumour development was not found. 2003 Elsevier Ltd. All rights reserved. 389. Respiratory hypersensitivity to trimellitic anhydride in Brown Norway rats: Analysis of dose-response following topical induction and time course following repeated inhalation challenge - Pauluhn J. [J. Pauluhn, Institute of Toxicology, BAYER HealthCare, Building No. 514, 42096 Wuppertal, Germany] TOXICOLOGY 2003 194 1-2 ; - summ in ENGL Trimellitic anhydride TMA ; is a low-molecular-weight chemical known to cause occupational asthma. The dose-response study was designed to determine whether respiratory responses during a single inhalation challenge with TMA 25-30mg m3 for 30 min, 3 weeks after the initial induction ; , the ensuing non-specific airway hyperresponsiveness AH ; to methacholine MCh ; aerosol, and infiltration of eosinophilic granulocytes into the lungs of sensitized Brown Norway BN ; rats are associated and dependent on the concentration of TMA used for topical induction. The initial topical exposure concentrations were 1, 5, and 25% TMA in acetone: olive oil AOO ; followed by a booster induction 1 week later. In the time course study BN rats received AOO alone or were sensitized to the minimal sensitizing topical concentration of TMA 5% ; and were the subsequently challenged with TMA on Days 17, 24, 41, and 66, followed by a MCh challenge 1 day later. One additional group of rats was sensitized to 5% TMA but were repeatedly challenged with MCh without prior TMA challenge. In the dose-response study the rats sensitized topically to TMA 5 and 25% in AOO ; displayed unequivocal changes in breathing patterns upon challenge with TMA, including an increased responsiveness to MCh aerosol. These findings were associated with a sustained pulmonary eosinophilic inflammation. All endpoints demonstrated consistently that 5% TMA in AOO constitutes the minimal sensitizing concentration. When rats were topically sensitized with this concentration and repeatedly challenged with TMA over a time period of 7 weeks, it became apparent that challenge exposures in BN rats may be false negative when performed at time periods less than 3 weeks after the initial induction. Despite the time-related increased responsiveness elicited by the repeated TMA challenge exposures, the MCh challenge revealed increased non-specific airway hyperreactivity exclusively on Day 17. After the sixth TMA-challenge, the respiratory response and lung weights of rats sensitized topically were essentially similar to those observed in the repetitively rechallenged control group induction: vehicle only; repeated booster challenge exposures with TMA ; . Thus, it appears, that in this animal model the effective concentration for successful topical sensitization must be at least 5%. The repeated low-dose re-challenge with TMA in topically sensitized rats resulted in similar or slightly aggravated time-related responses over a period of 7 weeks. An over-proportionally increased susceptibility of rats receiving a topical priming dose prior to repeated inhalation challenge exposures was not observed. In summary, this study shows that the analysis of Section 52 vol 43.2.
Note: For a description of references and other information, refer to the explanation of Committee tables and the accompanying notes at the end of this table. Footnotes: * Partially confirmed by bank information sources 10-14 ; * Fully confirmed by bank information sources 10-14 ; 1. Side agreement with Government of Iraq. 2. Ministry correspondence documents. 3. Company correspondence documents. 4. Other documents. 5. Ministry financial data. 6. Projected ASSF levied based on Government of Iraq policy documents. 7. Projected ASSF paid based on Government of Iraq policy documents. Represents contracts where inland transportation fee was required but no specific information was available 8. Projected Inland Transportation fees based on Government of Iraq policy documents. 9. Amount based on information provided by company and ministry documents. 10. Housing Bank for Trade and Finance Jordan ; , Central Bank of Iraq accounts Jan. 1, 2001 to Dec. 31, 2003 ; . 11. Jordan National Bank Jordan ; , Alia Company for Transport and General Trade accounts Mar. 1, 2000 to Dec. 31, 2003 ; . 12. Al-Rafidain Bank Jordan ; , Central Bank of Iraq accounts Jan. 1, 2000 to May 15, 2003 ; . 13. Fransabank SAL Lebanon ; , Central Bank of Iraq accounts Nov. 12, 2002 to Dec. 19, 2002 ; . 14. Jordan National Bank Jordan ; , Arrow Trans Shipping Company accounts May 1, 2001 to Dec. 31, 2001 ; . Page 375 of 381 and tenormin.
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24. Iwasaki, A., and R. Medzhitov. 2004. Toll-like receptor control of the adaptive immune responses. Nat. Immunol. 5: 987995. 25. Jakob, T., P. S. Walker, A. M. Krieg, E. von Stebut, M. C. Udey, and J. C. Vogel. 1999. Bacterial DNA and CpG-containing oligodeoxynucleotides activate cutaneous dendritic cells and induce IL-12 production: implications for the augmentation of Th1 responses. Int. Arch. Allergy Immunol. 118: 457461. 26. Johnson, D. H., and B. A. Cunha. 1993. Atypical pneumonias. Clinical and extrapulmonary features of Chlamydia, Mycoplasma, and Legionella infections. Postgrad. Med. 93: 6972, 7576, Kikuchi, T., T. Kobayashi, K. Gomi, T. Suzuki, Y. Tokue, A. Watanabe, and T. Nukiwa. 2004. Dendritic cells pulsed with live and dead Legionella pneumophila elicit distinct immune responses. J. Immunol. 172: 17271734. 28. Klein, T. W., C. Newton, R. Widen, and H. Friedman. 1993. Delta 9-tetrahydrocannabinol injection induces cytokine-mediated mortality of mice infected with Legionella pneumophila. J. Pharmacol. Exp. Ther. 267: 635640. 29. Klein, T. W., C. A. Newton, N. Nakachi, and H. Friedman. 2000. 9-Tetrahydrocannabinol treatment suppresses immunity and early IFN , IL-12, and IL-12 receptor 2 responses to Legionella pneumophila infection. J. Immunol. 164: 64616466. 30. Lu, T., C. Newton, I. Perkins, H. Friedman, and T. W. Klein. 2006. Role of cannabinoid receptors in delta-9-tetrahydrocannabinol suppression of IL12p40 in mouse bone marrow-derived dendritic cells infected with Legionella pneumophila. Eur. J. Pharmacol. 532: 170177. 31. Molofsky, A. B., B. G. Byrne, N. N. Whitfield, C. A. Madigan, E. T. Fuse, K. Tateda, and M. S. Swanson. 2006. Cytosolic recognition of flagellin by mouse macrophages restricts Legionella pneumophila infection. J. Exp. Med. 203: 10931104. 32. Napolitani, G., A. Rinaldi, F. Bertoni, F. Sallusto, and A. Lanzavecchia. 2005. Selected Toll-like receptor agonist combinations synergistically trigger a T helper type 1-polarizing program in dendritic cells. Nat. Immunol. 6: 769776. 33. Neild, A., T. Murata, and C. R. Roy. 2005. Processing and major histocompatibility complex class II presentation of Legionella pneumophila antigens by infected macrophages. Infect. Immun. 73: 23362343. 34. Neild, A. L., C. R. Roy, and E. R. Unanue. 2003. Legionella reveal dendritic cell functions that facilitate selection of antigens for MHC class II presentation. Immunity 18: 813823. 35. Newton, C., S. McHugh, R. Widen, N. Nakachi, T. Klein, and H. Friedman. 2000. Induction of interleukin-4 IL-4 ; by Legionella pneumophila infection in BALB c mice and regulation of tumor necrosis factor alpha, IL-6, and IL-1beta. Infect. Immun. 68: 52345240. 36. Newton, C. A., T. W. Klein, and H. Friedman. 1994. Secondary immunity to Legionella pneumophila and Th1 activity are suppressed by delta-9-tetrahydrocannabinol injection. Infect. Immun. 62: 40154020. 37. Palsson-McDermott, E. M., and L. A. O'Neill. 2004. Signal transduction by the lipopolysaccharide receptor, Toll-like receptor-4. Immunology 113: 153 162. Ren, T., D. S. Zamboni, C. R. Roy, W. F. Dietrich, and R. E. Vance. 2006. Flagellin-deficient Legionella mutants evade caspase-1- and Naip5-mediated macrophage immunity. PLoS Pathog. 2: e18. 39. Rutz, M., J. Metzger, T. Gellert, P. Luppa, G. B. Lipford, H. Wagner, and S. Bauer. 2004. Toll-like receptor 9 binds single-stranded CpG-DNA in a sequence- and pH-dependent manner. Eur. J. Immunol. 34: 25412550. 40. Salins, S., C. Newton, R. Widen, T. W. Klein, and H. Friedman. 2001. Differential induction of gamma interferon in Legionella pneumophila-infected macrophages from BALB c and A J mice. Infect. Immun. 69: 36053610. 41. Stunz, L. L., P. Lenert, D. Peckham, A. K. Yi, S. Haxhinasto, M. Chang, A. M. Krieg, and R. F. Ashman. 2002. Inhibitory oligonucleotides specifically block effects of stimulatory CpG oligonucleotides in B cells. Eur. J. Immunol. 32: 12121222. 42. Ulevitch, R. J., J. C. Mathison, and J. da Silva Correiaqq. 2004. Innate immune responses during infection. Vaccine 22 Suppl. 1 ; : S25S30. 43. Yamamoto, Y., T. W. Klein, C. A. Newton, R. Widen, and H. Friedman. 1988. Growth of Legionella pneumophila in thioglycolate-elicited peritoneal macrophages from A J mice. Infect. Immun. 56: 370375. 44. Yoshida, S., Y. Goto, Y. Mizuguchi, K. Nomoto, and E. Skamene. 1991. Genetic control of natural resistance in mouse macrophages regulating intracellular Legionella pneumophila multiplication in vitro. Infect. Immun. 59: 428432. 45. Zamboni, D. S., K. S. Kobayashi, T. Kohlsdorf, Y. Ogura, E. M. Long, R. E. Vance, K. Kuida, S. Mariathasan, V. M. Dixit, R. A. Flavell, W. F. Dietrich, and C. R. Roy. 2006. The Birc1e cytosolic pattern-recognition receptor contributes to the detection and control of Legionella pneumophila infection. Nat. Immunol. 7: 318325. 46. Zou, W., A. Amcheslavsky, and Z. Bar-Shavit. 2003. CpG oligodeoxynucleotides modulate the osteoclastogenic activity of osteoblasts via Toll-like receptor 9. J. Biol. Chem. 278: 1673216740.
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The 50 most advertised drugs amassed retail sales of $41.3 billion in 2000, increasing at 2.3 times the rate of all other drugs on the market. National Institute for Health Care Management, "Prescription Drugs and Mass Media Advertising: 1999-2000, " November 2001 ; In a survey of physicians in 1998, 97% of allergists said their patients were influenced by DTC advertising. Barents Group LLC, "Factors Affecting the Growth of Prescription Drug Expenditures, " National Institute for Health Care Management Research and Educational Foundation, 1999 ; Nearly 1 out of every 3 adults has talked to a doctor and 1 in 8 has received a prescription drug in response to an advertisement. Kaiser Family Foundation, "Understanding the Effects of Direct-to-Consumer Prescription Drug Advertising, " November 2001 ; Advertising is dominated by drugs that are not life-saving: Four of the five prescription drugs most advertised in 1999 were so-called "lifestyle" drugs: Claritin antihistamine ; $136.8 million, Xenical anti-obesity ; $76.2 million, Peopecia male pattern baldness ; $71.1 million, Zyrtec antihistamine ; $57.1 million. National Institute for Health Care Management, "Prescription Drugs and Mass Media Advertising, " September 2000 ; Advertising is becoming more important to drug companies: The drug industry is shifting the core of its business away from the often unpredictable task of creating drugs and toward the steadier business of marketing them. Harris, "Drug Firms, Stymied in the Lab, Become Marketing Machines, " The Wall Street Journal, July 6, 2000 ; "The industry is pinning its growth hopes less on new products and more on persuading people, including healthy ones, to buy the pills already being sold." Marketing of Viagra to healthy young men is an example. Harris, "Drug Firms, Stymied in the Lab, Become Marketing Machines, " The Wall Street Journal, July 6, 2000 ; Prescription drug advertising is not always accurate: Of the estimated 200 different drug commercials aired on TV since the FDA relaxed rules on advertising in 1997, the FDA has issued 45.
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PURSUANT TO THE TEXAS CONTROLLED SUBSTANCES ACT, HEALTH AND SAFETY CODE, CHAPTER 481, THESE SCHEDULES, ESTABLISHED JANUARY 1, 1998, SUPERCEDE PREVIOUS SCHEDULES AND CONTAIN THE MOST CURRENT VERSION OF THE SCHEDULES OF ALL CONTROLLED SUBSTANCES FROM THE PREVIOUS SCHEDULES AND MODIFICATIONS. January 1, 1998 Changes to the schedules are designated by an asterisk * ; . Additional information can be obtained by contacting the Texas Department of Health, Bureau of Food and Drug Safety, 1100 West 49th Street, Austin, Texas 78756. The telephone number is 512 ; 719-0237. SCHEDULES Nomenclature: Controlled substances listed in these schedules are included by whatever official, common, usual, chemical, or trade name they may be designated. SCHEDULE I Schedule I consists of: ! Schedule I opiates the following opiates, including their isomers, esters, ethers, salts, and salts of isomers, esters, and ethers, unless specifically excepted, if the existence of these isomers, esters, ethers, and salts is possible within the specific chemical designation: 1 ; Acetyl-alpha-methylfentanyl N-[1- 1-methyl-2-phenethyl ; -4-piperidinyl]-Nphenylacetamide 2 ; Allylprodine; 3 ; Alphacetylmethadol except levo-alphacetylmethadol, also known as levo-alphaacetylmethadol, levomethadyl acetate, or LAAM 4 ; Alpha-methylfentanyl or any other derivative of Fentanyl; 5 ; Alpha-methylthiofentanyl N-[1-methyl-2- 2-thienyl ; 6 ; Benzethidine; 7 ; Beta-hydroxyfentanyl N-[1- 2-hydroxy-2-phenethyl ; 8 ; Beta-hydroxy-3-methylfentanyl N-[1- 2-hydroxy-2-phenethyl ; -3-methyl4-piperidinyl]- N-phenylpropanamide 1 and zovirax.
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